Learn about the functional medicine approach to thyroid health and its role in supporting your body’s needs.
Hello, I’m Dr. Alex Jimenez. Welcome to our educational post where we delve into the intricate world of thyroid health. For many individuals, symptoms like persistent fatigue, weight gain, brain fog, and feeling cold are quickly attributed to a thyroid problem, often leading to a prescription for medications like Synthroid or levothyroxine. However, this approach may not address the root cause of the dysfunction. In this comprehensive guide, we will explore the complex journey of thyroid hormones, moving beyond the thyroid gland itself to understand its profound connection with the liver, gut, and adrenal system. I’ll share the latest findings from leading researchers through the lens of evidence-based functional medicine. We will uncover the four primary physiological mechanisms that account for over 90% of thyroid-related issues—none of which originate within the thyroid gland itself. These mechanisms involve systemic inflammation, liver congestion, gut dysbiosis, and chronic stress responses.
I will use a real-world case study to illustrate how these systems can go awry and how a functional medicine approach can restore balance without lifelong medication. We will also discuss the vital role of our integrated clinical model at Injury Medical Clinic. Here, my work in chiropractic and functional medicine is complemented by the medical oversight of Dr. Maria Guadalupe Cardenas, MD, our experienced Medical Director. This collaborative, multidisciplinary setup allows us to offer a holistic and comprehensive treatment path that integrates chiropractic adjustments, functional nutrition, rehabilitation, and medical management to address the underlying causes of chronic health conditions, including thyroid dysfunction. Join me on this educational journey to empower yourself with knowledge and discover a path toward true metabolic healing.
Before we dive into the complexities of thyroid physiology, I believe it’s essential to provide some context about our unique clinical environment here at Injury Medical Clinic PA in El Paso, Texas. Our practice is built on a foundation of integrative and multidisciplinary care. I am Dr. Alex Jimenez, and I hold several qualifications, including Doctor of Chiropractic (DC), Advanced Practice Registered Nurse (APRN), and certifications as a Family Nurse Practitioner (FNP-BC) and Functional Medicine Certified Practitioner (IFMCP). This diverse training allows me to view patient health through multiple lenses, from the biomechanical and neurological aspects of chiropractic to the systemic, evidence-based approach of functional medicine.
However, our clinic’s strength lies in collaboration. I have the distinct privilege of working alongside Dr. Maria Guadalupe Cardenas, MD. Dr. Cardenas is a board-certified internist with an impressive career spanning over 40 years. Her extensive experience provides invaluable medical expertise and oversight to our practice. She serves as our Medical Director and Collaborative Physician, a structure common in progressive integrative care settings. This partnership ensures that our patients receive a truly comprehensive evaluation and treatment plan.
This model allows us to integrate various disciplines under one roof seamlessly:
This synergistic model means that when a patient like Jennifer (our case study for today) comes to us, she isn’t just seeing a chiropractor or a medical doctor in isolation. She is benefiting from a team that communicates and strategizes together. We can address the structural misalignments contributing to her stress response with chiropractic adjustments, use functional medicine to heal her gut and liver, and have the medical oversight of Dr. Cardenas to guide her journey, especially when tapering off medications safely. This powerful combination allows us to unravel complex cases and guide our patients back to vibrant health.
Let me start with a story that is all too common in my thirty years of practice. It’s the story of Jennifer, a 41-year-old woman who reached out to me in complete desperation. She sent me a video message, and through her tears, she painted a picture of a life that had been stolen from her. For seven long years, Jennifer had been doing everything her doctors told her to do. She had been diagnosed with hypothyroidism and was taking not one, but two different thyroid medications—Synthroid (levothyroxine sodium) and another form of levothyroxine. Yet, instead of feeling better, her health was spiraling downward.
She was gaining weight despite her efforts, her hands and feet were perpetually cold, and a thick brain fog clouded her thoughts, making it hard to focus or feel like herself. A deep, persistent depression had settled over her, and she described her daily existence as “dragging herself through life.” She had consulted five different doctors over those seven years, and each time, the answer was the same: adjust the medication. But the medication wasn’t working. In fact, she felt it was making things worse.
Jennifer’s story powerfully illustrates a fundamental misunderstanding in the conventional treatment of thyroid disorders. Her experience highlights a critical gap in care: focusing on replacing a hormone without asking why the body can’t use it effectively in the first place. This is where functional medicine offers a beacon of hope. It prompts us to look deeper, beyond the thyroid gland itself, and into the intricate web of systems that govern our metabolic health. Her journey inspired me to write this post and share the knowledge that there is often another way—a way to reclaim your health by addressing the root cause.
To understand why Jennifer’s treatment was failing, we need to go back to biology 101 and review how the thyroid system actually works. It’s a common misconception that the thyroid gland runs the whole show. In reality, it’s more like a specialized factory with a very specific job.
Think of the thyroid gland, that small, butterfly-shaped organ in your neck, as a manufacturing plant. Its primary role, under the direction of the brain (specifically the pituitary gland, which releases Thyroid Stimulating Hormone, or TSH), is to produce a storage hormone called thyroxine, or T4. That’s essentially it. It cranks out T4, which is largely inactive. It’s a prohormone, meaning it’s a precursor that needs to be converted into something else to have a powerful effect. The thyroid gland does produce a very small amount of the active hormone, T3, but the vast majority of its output is T4.
So, if the thyroid gland only makes the storage hormone, where does the active, energy-driving hormone come from? This is the crucial part of the story that is so often overlooked. The conversion of inactive T4 into the biologically active triiodothyronine (T3) doesn’t primarily happen in the thyroid. It happens in other tissues throughout the body, which I call the “conversion engines.”
These key conversion sites include:
This process of converting T4 to T3 is called peripheral conversion. If this conversion process is broken or inefficient, you will experience all the classic symptoms of hypothyroidism—fatigue, weight gain, depression, cold intolerance—because your cells are literally being starved of the active hormone they need to function. You are, in essence, running on an empty metabolic tank.
Now, let’s follow up with Jennifer. Her doctors were giving her more and more T4 medication (Synthroid, levothyroxine). Some physicians might even use Armor Thyroid or other desiccated forms, which contain both T4 and T3. But here’s the problem: if the body’s conversion engines are broken, pouring more raw material (T4) into the system is not only ineffective, but it can also be detrimental.
When the body is under stress, whether from inflammation, nutrient deficiencies, or toxins, it has a protective mechanism. It shunts the conversion of T4 down a different pathway. Instead of making active T3, it starts producing a hormone called Reverse T3 (rT3). Reverse T3 is an isomer of T3, meaning it has the same atoms but a different structural arrangement. This seemingly small difference renders it biologically inactive. More than that, rT3 acts as a competitive inhibitor. It fits perfectly into the T3 receptors on our cells, but it doesn’t activate them. It’s like putting the wrong key into a lock; it gets stuck and prevents the right key (active T3) from getting in and doing its job.
So, for someone like Jennifer, whose peripheral conversion was clearly impaired, taking high doses of T4 medication was providing more substrate for her stressed body to convert into rT3. This not only failed to resolve her symptoms but actively made them worse by increasing the amount of this “metabolic brake” in her system. All her biology was doing was converting those medications into reverse T3, exacerbating her fatigue, weight gain, and brain fog. Her experience is a classic case of what we see when the cause of thyroid dysfunction isn’t in the thyroid gland itself, but in the peripheral tissues responsible for hormone activation.
In my three decades of clinical practice focusing on functional medicine, I have consistently observed that over 90% of thyroid problems can be traced back to four primary physiological mechanisms. The beauty of this framework is that none of them originate in the thyroid gland itself; therefore, none fundamentally require medication as the first or only line of treatment. These mechanisms are detectable through proper testing, explainable through established biochemistry, and addressable through targeted, non-pharmacological interventions. Let’s break them down one by one.
The first and perhaps most pervasive driver of thyroid dysfunction is systemic inflammation. This isn’t the acute, localized inflammation you feel when you sprain an ankle. This is chronic, low-grade inflammation that simmers throughout the body, disrupting normal physiological processes. The sources of this inflammation are varied and can include:
Regardless of the source, chronic inflammation results in the body “dumping” large quantities of inflammatory messengers called cytokines into circulation. Molecules like Tumor Necrosis Factor-alpha (TNF-α), Interleukin-6 (IL-6), and Interleukin-1-beta (IL-1β) are like biological grenades. They are designed to coordinate an immune attack but cause significant collateral damage when they are chronically elevated.
One of their primary targets is the delicate enzymatic machinery responsible for thyroid hormone conversion. Here’s the specific biochemical cascade:
So, the net effect of systemic inflammation is a perfect storm for thyroid dysfunction: it actively blocks the conversion of T4 to active T3 while simultaneously shunting T4 toward the production of the inhibitory rT3. This is a survival mechanism; in times of severe illness or injury (which the body perceives as inflammation), it makes sense to slow down metabolism to conserve energy for healing and immune function. However, in the context of chronic, low-grade inflammation, this adaptive response becomes maladaptive, leading to the debilitating symptoms of hypothyroidism despite normal or even high levels of T4.
The liver is the unsung hero of endocrine health, and its role in thyroid metabolism is absolutely central. As I mentioned, up to 80% of T4-to-T3 conversion occurs inside the liver’s cells, the hepatocytes, through the action of the D1 deiodinase enzyme. When the liver is not healthy, this entire process grinds to a halt.
One of the most common forms of liver dysfunction in the modern world is Non-Alcoholic Fatty Liver Disease (NAFLD). Often driven by a diet high in processed carbohydrates, sugar, and industrial seed oils, this condition causes hepatocytes to accumulate lipid droplets. This accumulation doesn’t just make the liver “fatty”; it induces a state of cellular stress known as endoplasmic reticulum (ER) stress. The endoplasmic reticulum is the part of the cell responsible for folding proteins into their correct functional shapes. When it’s stressed by lipid overload and inflammation, it can’t do its job properly.
The D1 deiodinase enzyme is a protein that must be correctly folded to function. Under ER stress, D1 deiodinase production and activity plummet. The conversion engine inside the liver effectively shuts down.
But the liver’s role doesn’t stop at conversion. It’s also responsible for conjugating thyroid hormones. This detoxification process attaches a molecule (like glucuronic acid or a sulfate group) to used hormones, making them water-soluble so they can be excreted from the body, primarily through bile into the intestines.
This process is also crucial for recycling. Some of the conjugated T3 excreted in bile can be “deconjugated” by healthy gut bacteria and reabsorbed back into circulation, a process known as the enterohepatic loop. This is an elegant system for conserving and recycling valuable hormones.
However, in a congested or “fatty” liver, the quality of bile itself changes. It becomes thick, viscous, and sludgy. This sluggish bile flow, or cholestasis, causes a major traffic jam.
A congested liver therefore delivers a double blow to the thyroid system: it fails to convert the inactive hormone to the active form. It also fails to clear hormonal waste products, leading to a toxic, low-energy internal environment. Addressing liver health is non-negotiable for restoring thyroid function.
The gut’s contribution to thyroid health is often underestimated. While it accounts for a seemingly modest 20% of T4-to-T3 conversion, its influence extends far beyond that, affecting the liver, the immune system, and the body’s overall inflammatory status.
As mentioned in the discussion of the liver, conjugated T3 is excreted via bile into the intestinal tract. Here, a healthy, diverse gut microbiome plays a critical enzymatic role. Certain species of beneficial bacteria produce an enzyme called beta-glucuronidase. This enzyme acts like a pair of molecular scissors, snipping off the glucuronic acid molecule that the liver attached to the T3. This “deconjugation” process liberates the T3, allowing it to be reabsorbed from the gut back into the portal circulation, which leads directly to the liver. This recycling pathway ensures that the body retains and reuses its active thyroid hormone efficiently.
Now, consider what happens in dysbiosis, an imbalance in the gut microbiome where pathogenic or opportunistic bacteria overgrow and beneficial species are diminished. The bacteria that produce beta-glucuronidase are gone. As a result, the conjugated T3 that enters the gut is never recycled. It remains bound and is flushed right down the toilet in your stool. This represents a significant, continuous loss of active thyroid hormone, forcing the body to constantly produce and convert more and placing a huge strain on the entire system.
The consequences of an unhealthy gut go even further. Dysbiosis is almost always accompanied by increased intestinal permeability, commonly known as “leaky gut.” The single-cell layer lining our intestines is meant to be a tightly controlled barrier, allowing digested nutrients to pass through while keeping bacteria, undigested food particles, and toxins out. In a leaky gut, the tight junctions between these cells become loose.
This allows highly inflammatory components of bacterial cell walls, specifically lipopolysaccharides (LPS), to leak from the gut into the systemic circulation. LPS, also known as endotoxin, is one of the most potent triggers of inflammation known to our immune system. When immune cells detect LPS in the bloodstream—where it absolutely does not belong—they mount a massive defensive response, cranking up the production of the very same inflammatory cytokines (TNF-α, IL-6) that we discussed earlier.
This creates a direct link between gut health and thyroid conversion in the liver. LPS leaking from the gut travels through the portal vein directly to the liver, where it triggers intense inflammation, annihilating D1 deiodinase activity and shutting down T4-to-T3 conversion at its primary site. This is how a gut problem can directly manifest as thyroid symptoms. Healing the gut by removing inflammatory triggers, restoring microbial balance, and repairing the gut lining is therefore a foundational step in any thyroid recovery protocol.
This final mechanism is where I see so many well-intentioned people get it wrong. The body’s stress response system, the Hypothalamic-Pituitary-Adrenal (HPA) axis, is designed for short-term survival. Physiologically, your biology cannot tell the difference between the acute stress of being chased by a predator, the chronic psychological stress of a demanding job or difficult relationship, or the physiological stress of a prolonged caloric deficit and intense exercise. To the HPA axis, they all look identical: a threat to survival.
In response to any of these stressors, the HPA axis fires up, culminating in the release of the stress hormone cortisol from the adrenal glands. Cortisol is catabolic; its job is to mobilize energy (by breaking down muscle and fat) to fuel a “fight or flight” response. As part of this energy-conservation strategy, it also aims to slow non-essential, long-term metabolic processes.
One of its primary targets is thyroid metabolism. Elevated cortisol directly crushes the activity of the D1 deiodinase enzyme in the liver and other peripheral tissues. Just like inflammation, it powerfully inhibits the conversion of T4 to T3. This makes perfect evolutionary sense. If you are in a famine or running for your life, the last thing your body needs is a roaring-hot metabolism burning through precious energy reserves. It needs to slow down, conserve, and survive.
Herein lies the trap. A person feels tired, notices they’ve gained weight, and correctly deduces their metabolism is slow. They decide to “fix” it. What do they do? They start an aggressive diet, perhaps incorporating intermittent fasting, and dramatically drop their calories into a huge caloric deficit. They pair this with high-intensity workouts. They have great commitment, but from a biochemical perspective, the timing is terrible.
Let’s look at what this does to the body, especially the liver:
The liver interprets this combination of low glycogen, low insulin, and low leptin as an unequivocal signal of famine. Its prime directive becomes survival. It immediately shuts down D1 deiodinase activity to conserve energy. The person’s attempt to “boost” their metabolism has paradoxically slammed the brakes even harder, worsening thyroid symptoms and making weight loss nearly impossible. This is why we must always address the HPA axis and ensure the body feels “safe” before attempting any metabolic push. Restoring a sense of safety through stress management, adequate nutrition, and nervous system regulation is paramount.
At this point, you might be wondering, “This is all fascinating biochemistry, but where does chiropractic fit in?” This is an excellent question, and the answer lies in the profound connection between the spine, the nervous system, and the body’s ability to regulate stress and inflammation—two of the key drivers of thyroid dysfunction we’ve just discussed.
As a Doctor of Chiropractic, my primary focus is detecting and correcting vertebral subluxations. A subluxation is a condition where a spinal vertebra has lost its normal position or motion, interfering with the nerve signals that travel between the brain and the body. This neurological interference doesn’t just cause back pain or neck stiffness; it creates a state of background static or stress in the nervous system, a condition known as dysafferentation.
The nervous system is the master controller of the HPA axis. The brain perceives threats and initiates the stress response based on the total of information it receives from the body. When there are subluxations, particularly in the upper cervical (neck) and upper thoracic (mid-back) regions, the altered biomechanics and nerve signaling can be interpreted by the brain as a persistent, low-level threat. This can keep the HPA axis chronically activated, leading to elevated cortisol levels.
Research shows that chiropractic adjustments can directly and measurably affect the body’s stress physiology. By restoring proper motion and alignment to the spine, an adjustment reduces the aberrant neurological input (dysafferentation) to the brain. This can help to “down-regulate” the sympathetic “fight or flight” nervous system and promote a shift toward the parasympathetic “rest and digest” state.
Studies have demonstrated that chiropractic care can:
For a patient like Jennifer, whose body is clearly locked in a state of chronic stress, integrative chiropractic care becomes an essential component of her recovery. It’s a powerful, non-invasive tool to help her body exit the survival mode that is preventing her from healing. It helps turn off the “cortisol crush” and quell the fires of inflammation, creating the physiological space her liver, gut, and other tissues need to begin functioning properly again. This is a perfect example of how our clinic’s collaborative approach—combining Dr. Cardenas’s medical insight with my chiropractic and functional medicine expertise—allows us to address the patient’s health from multiple, synergistic angles.
The unifying thread among these four mechanisms—inflammation, liver congestion, gut dysbiosis, and HPA axis dysfunction—is that they are detectable, explainable, and, most importantly, addressable without a single prescription being written. The entire playbook for diagnosing and correcting these issues is well documented in scientific and clinical research.
The first step is always proper testing. A standard thyroid panel (TSH and maybe Total T4) is woefully inadequate. To get a complete picture, we need to look at:
These labs provide the data we need to pinpoint exactly where the breakdown is occurring. Is it inflammation from a latent virus? Is it LPS from a leaky gut shutting down the liver? Is it a cortisol and rT3 issue from chronic stress and undereating? For Jennifer, her labs (which I will be sharing in my stories on our social platforms) revealed a classic pattern: high reverse T3, low-normal free T3, elevated inflammatory markers, and signs of HPA axis dysregulation.
Armed with this data, we can build a personalized treatment protocol that addresses the root cause(s). This is not a one-size-fits-all approach. It may include:
This comprehensive, systems-based approach is how we guide patients like Jennifer back to health. It’s not a quick fix, but it is a true fix. By restoring function to the body’s core systems, we create an environment where the thyroid can do its job and where thyroid hormones—whether produced by the body or supplemented temporarily—can be used effectively. This is the promise and the power of functional medicine, and it is a journey I am honored to guide my patients on every day. Thank you for joining me.
Creation Date: August 26, 2026
Time to Read: Approximately 25 minutes.
SEO Tags: thyroid health, functional medicine, hypothyroidism, Dr. Alex Jimenez, Dr. Maria Guadalupe Cardenas, integrative care, El Paso Texas, chiropractic care, Hashimoto’s, reverse T3, liver function, gut health, HPA axis, systemic inflammation, cortisol, T4 to T3 conversion, deiodinase, leaky gut, dysbiosis, Synthroid, levothyroxine, brain fog, weight gain, fatigue, holistic health, vertebral subluxation, personal injury clinic, rehabilitation, evidence-based research, functional nutrition
Professional Scope of Practice *
The information herein on "Thyroid Health Strategies in a Functional Medicine Approach" is not intended to replace a one-on-one relationship with a qualified health care professional or licensed physician and is not medical advice. We encourage you to make healthcare decisions based on your research and partnership with a qualified healthcare professional.
Blog Information & Scope Discussions
Welcome to El Paso's Premier Fitness, Injury Care Clinic & Wellness Blog, where Dr. Alex Jimenez, DC, FNP-C, a Multi-State board-certified Family Practice Nurse Practitioner (FNP-BC) and Chiropractor (DC), presents insights on how our multidisciplinary team is dedicated to holistic healing and personalized care. Our practice aligns with evidence-based treatment protocols inspired by integrative medicine principles, similar to those found on this site and our family practice-based chiromed.com site, focusing on restoring health naturally for patients of all ages.
Our areas of multidisciplinary practice include Wellness & Nutrition, Chronic Pain, Personal Injury, Auto Accident Care, Work Injuries, Back Injury, Low Back Pain, Neck Pain, Migraine Headaches, Sports Injuries, Severe Sciatica, Scoliosis, Complex Herniated Discs, Fibromyalgia, Chronic Pain, Complex Injuries, Stress Management, Functional Medicine Treatments, and in-scope care protocols.
Our information scope is multidisciplinary, focusing on musculoskeletal and physical medicine, wellness, contributing etiological viscerosomatic disturbances within clinical presentations, associated somato-visceral reflex clinical dynamics, subluxation complexes, sensitive health issues, and functional medicine articles, topics, and discussions.
We provide and present clinical collaboration with specialists from various disciplines. Each specialist is governed by their professional scope of practice and their jurisdiction of licensure. We use functional health & wellness protocols to treat and support care for musculoskeletal injuries or disorders.
Our videos, posts, topics, and insights address clinical matters and issues that are directly or indirectly related to our clinical scope of practice.
Our office has made a reasonable effort to provide supportive citations and has identified relevant research studies that support our posts. We provide copies of supporting research studies upon request to regulatory boards and the public.
We understand that we cover matters that require an additional explanation of how they may assist in a particular care plan or treatment protocol; therefore, to discuss the subject matter above further, please feel free to ask Dr. Alex Jimenez, DC, APRN, FNP-BC, or contact us at 915-850-0900.
We are here to help you and your family.
Blessings
Dr. Alex Jimenez DC, MSACP, APRN, FNP-BC*, CCST, IFMCP, CFMP, ATN
email: coach@elpasofunctionalmedicine.com
Multidisciplinary Licensing & Board Certifications:
Licensed as a Doctor of Chiropractic (DC) in Texas & New Mexico*
Texas DC License #: TX5807, Verified: TX5807
New Mexico DC License #: NM-DC2182, Verified: NM-DC2182
Multi-State Advanced Practice Registered Nurse (APRN*) in Texas & Multi-States
Multistate Compact APRN License by Endorsement (42 States)
Texas APRN License #: 1191402, Verified: 1191402 *
Florida APRN License #: 11043890, Verified: APRN11043890 *
Verify Link: Nursys License Verifier
* Prescriptive Authority Authorized
ANCC FNP-BC: Board Certified Nurse Practitioner*
Compact Status: Multi-State License: Authorized to Practice in 40 States*
Graduate with Honors: ICHS: MSN-FNP (Family Nurse Practitioner Program)
Degree Granted. Master's in Family Practice MSN Diploma (Cum Laude)
Dr. Alex Jimenez, DC, APRN, FNP-BC*, CFMP, IFMCP, ATN, CCST
(Board Certified: Family Practice Nurse Practitioner—Multistate)*
(Licensed Nurse Practitioner & Chiropractor - Multistate)*
Clinical Director
Digital Business Card
Dr. Maria Cardenas, MD
(Board Certified: Internal Medicine)
(Licensed Medical Doctor)
Medical Director, Clinical Director & Collaborative Physician
NPI # 1164426748
MD License #: J2933
Licenses and Board Certifications:
MD: Medical Doctor
DC: Doctor of Chiropractic
APRNP: Advanced Practice Registered Nurse
FNP-BC: Family Practice Specialization (Multi-State Board Certified)
RN: Registered Nurse (Multi-State Compact License)
CFMP: Certified Functional Medicine Provider
MSN-FNP: Master of Science in Family Practice Medicine
MSACP: Master of Science in Advanced Clinical Practice
IFMCP: Institute of Functional Medicine
CCST: Certified Chiropractic Spinal Trauma
ATN: Advanced Translational Neutrogenomics
Memberships & Associations:
TCA: Texas Chiropractic Association: Member ID: 104311
AANP: American Association of Nurse Practitioners: Member ID: 2198960
ANA: American Nurse Association: Member ID: 06458222 (District TX01)
TNA: Texas Nurse Association: Member ID: 06458222
NPI: 1205907805
| Primary Taxonomy | Selected Taxonomy | State | License Number |
|---|---|---|---|
| No | 111N00000X - Chiropractor | NM | DC2182 |
| Yes | 111N00000X - Chiropractor | TX | DC5807 |
| Yes | 363LF0000X - Nurse Practitioner - Family | TX | 1191402 |
| Yes | 363LF0000X - Nurse Practitioner - Family | FL | 11043890 |
| Yes | 363LF0000X - Nurse Practitioner - Family | CO | C-APN.0105610-C-NP |
| Yes | 363LF0000X - Nurse Practitioner - Family | NY | N25929 |
Dr. Alex Jimenez, DC, APRN, FNP-BC*, CFMP, IFMCP, ATN, CCST
(Board Certified: Family Practice Nurse Practitioner—Multistate)*
(Licensed Nurse Practitioner & Chiropractor - Multistate)*
Clinical Director
Digital Business Card
Dr. Maria Cardenas, MD
(Board Certified: Internal Medicine)*
(Licensed Medical Doctor)*
Medical Director, Clinical Director & Collaborative Physician
NPI # 1164426748
MD License #: J2933
Functional Nutrition for Neck and Brain Recovery Guide Abstract A head or neck injury can… Read More
By Dr. Alex Jimenez, DC, APRN, FNP-BC, CFMP, IFMCP, ATN, CCST Read More
Stop the Wait-and-See Loop After a Car Accident Abstract Conventional personal injury care often sends… Read More
By Dr. Alex Jimenez, DC, APRN, FNP-BC, CFMP, IFMCP, ATN, CCST Read More
Complex Spine Pain Second Opinion in El Paso, TX Abstract Complex spinal joint and nerve… Read More
Sports Neuropathies: How Integrative Chiropractic Care Helps Athletes Recover from Nerve Injuries Abstract Neuropathies do… Read More
Personal Injury, Trauma & Spine Rehab Specialists